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Publication
The Familiality of Rapid Renal Decline in Diabetes.
Authors
Frodsham SG, Yu Z, Lyons AM, Agarwal A, Pezzolesi MH, Dong L, Srinivas TR, Ying
J, Greene T, Raphael KL, Smith KR, Pezzolesi MG
Submitted By
Marcus Pezzolesi on 2/5/2019
Status
Published
Journal
Diabetes
Year
2019
Date Published
2/1/2019
Volume : Pages
68 : 420 - 429
PubMed Reference
Abstract
Sustained and rapid loss of glomerular filtration rate (GFR) is the predominant
clinical feature of diabetic kidney disease and a requisite for the development
of end-stage renal disease. Although GFR trajectories have been studied in
several cohorts with diabetes and without diabetes, whether rapid renal decline
clusters in families with diabetes has not been examined. To determine this, we
estimated GFR (eGFR) from serum creatinine measurements obtained from 15,612
patients with diabetes at the University of Utah Health Sciences Center and
established their renal function trajectories. Patients with rapid renal decline
(eGFR slope < -5 mL/min/1.73 m2/year) were then mapped to pedigrees using
extensive genealogical records from the Utah Population Database to identify
high-risk rapid renal decline pedigrees. We identified 2,127 (13.6%) rapid
decliners with a median eGFR slope of -8.0 mL/min/1.73 m2/year and 51 high-risk
pedigrees (ranging in size from 1,450 to 24,501 members) with excess clustering
of rapid renal decline. Familial analysis showed that rapid renal decline
aggregates in these families and is associated with its increased risk among
first-degree relatives. Further study of these families is necessary to
understand the magnitude of the influence of shared familial factors, including
environmental and genetic factors, on rapid renal decline in diabetes.
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Please acknowledge all posters, manuscripts or scientific materials that were generated in part or whole using funds from the Diabetic Complications Consortium(看片视频) using the following text:
Financial support for this work provided by the NIDDK Diabetic Complications Consortium (RRID:SCR_001415, www.diacomp.org), grants DK076169 and DK115255
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