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Publication
Genome-wide DNA methylation profiling of human diabetic peripheral neuropathy in
subjects with type 2 diabetes mellitus.
Authors
Guo K, Elzinga S, Eid S, Figueroa-Romero C, Hinder LM, Pacut C, Feldman EL, Hur
J
Submitted By
Junguk Hur on 10/14/2019
Status
Published
Journal
Epigenetics
Year
2019
Date Published
8/1/2019
Volume : Pages
14 : 766 - 779
PubMed Reference
Abstract
DNA methylation is an epigenetic mechanism important for the regulation of gene
expression, which plays a vital role in the interaction between genetic and
environmental factors. Aberrant epigenetic changes are implicated in the
pathogenesis of diabetes and diabetic complications, but the role of DNA
methylation in diabetic peripheral neuropathy (DPN) is not well understood.
Therefore, our aim in this study was to explore the role of DNA methylation in
the progression of DPN in type 2 diabetes. We compared genome-wide DNA
methylation profiles of human sural nerve biopsies from subjects with stable or
improving nerve fibre counts to biopsies from subjects with progressive loss of
nerve fibres. Nerve fibre counts were determined by comparing myelinated nerve
fibre densities between an initial and repeat biopsy separated by 52 weeks.
Subjects with significant nerve regeneration (regenerators) and subjects with
significant nerve degeneration (degenerators) represent the two extreme DPN
phenotypes. Using reduced representation bisulfite sequencing, we identified
3,460 differentially methylated CpG dinucleotides between the two groups. The
genes associated with differentially methylated CpGs were highly enriched in
biological processes that have previously been implicated in DPN such as nervous
system development, neuron development, and axon guidance, as well as
glycerophospholipid metabolism and mitogen-activated protein kinase (MAPK)
signalling. These findings are the first to provide a comprehensive analysis of
DNA methylation profiling in human sural nerves of subjects with DPN and suggest
that epigenetic regulation has an important role in the progression of this
prevalent diabetic complication.
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Please acknowledge all posters, manuscripts or scientific materials that were generated in part or whole using funds from the Diabetic Complications Consortium(看片视频) using the following text:
Financial support for this work provided by the NIDDK Diabetic Complications Consortium (RRID:SCR_001415, www.diacomp.org), grants DK076169 and DK115255
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